By Oliver Trust
BERLIN cheap nike air max 97 womens , June 26 (Xinhua) -- Germany has never failed to get past the group stage at the World Cup and this year is no exception. By beating the USA through a Thomas Mueller goal in the 55th minute, Joachim Loew's team has comfortably topped Group G in Brazil. Juergen Klinsmann's team, however, can count themselves lucky to have qualified for the last 16 as runners-up as Portugal's 2-1 win against Ghana was not enough for them to finish above the Americans.
Germany's head coach Joachim Loew changed his team by bringing in Bastian Schweinsteiger in central midfield and Lukas Podolski on the left wing for Mario Goetze. With Schweinsteiger in the thick of the action cheap nike air max 97 mens , Germany bossed play right from the kick-off. The three up front, Podolski, Thomas Mueller and Mesut Oezil were continuously changing positions, with Mueller moving continuously from the center to both flanks and back.
German had a lot of possession. Schweinsteiger and Kroos played slightly further up the pitch cheap nike air max 97 white , as did the German defense, to move into space quicker and stifle Michael Bradley, Clint Dempsey and US counterattacks.
Everything worked well in the first 15 minutes despite the atrocious conditions as it had rained for hours before the start of the game. The Germans were constantly changing the pace of the game to make the US defense work harder. In the first ten minutes alone, Mueller came close to scoring on three occasions but lacked the precision he showed in the first game against Portugal.
The final deadly pass was not coming off. Every now and again the transition from defense to attack was too slow. But Loew's team wasn't intent on taking too many risks but they nevertheless controlled play in the first half and made far fewer mistakes than in the Ghana match. Team USA found more space towards the end of the first half but were unable to capitalize.
Then Loew sent on Miroslav Klose for Podolski who was not able to break down the US defense. The substitution strengthened the spearhead and they became more dangerous when going forward. It didn't take long for Mueller to put Germany 1-0 up in the 55th minute and firmly in control of the group. If they were to finish second cheap nike air max 97 black , USA had to hope Ghana and Portugal would neutralize each other.
Despite being regularly fouled, Schweinsteiger, Germany's man of the match, stayed in control of the midfield and was instrumental in ensuring his team was never in any danger of losing the match. Juergen Klinsmann's side simply could not break down the German system. Based on a solid defense and midfield they closed the Americans down and at the same time provided the platform for quick incisive attacks. If they continue in the same vein they will be very difficult to beat in the knockout stages.
Not wanting to waste energy and risk injuries cheap nike air max 2018 womens , the solid Germans played within themselves and comfortably saw out the match. Schweinsteiger was the key figure, as long as his energy was top, but was backed up by Lahm and Kroos. Oezil was much improved and Mueller scored his fourth goal of the tournament.
And the German defense gave away nothing, right and left back had more space than in the Ghana game. The most important news for Germany's coach Loew was that his team rarely made any mistakes and that they found answers in every situation. The German game is clicking into place and they must be one of the tournament favorites.
With the Group H matches still to come cheap nike air max 2018 mens , Germany and the US will know later on Thursday evening which of Belgium, Russia and Algeria they will face in the last 16.
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SAN FRANCISCO, March 18 (Xinhua) -- An almost accidental discovery at a laboratory in Stanford University, California www.cheapairmax97shoes.com , has raised hopes of finding alternative therapies to cure a type of blood cancer by turning the dangerous cells into white blood cells, the "good guys."
These immune cells are also able to detect and destroy other cancer cells, therefore providing a fighting chance against one of the most aggressive blood cancers, B-cell acute lymphoblastic leukemia with a mutation called the Philadelphia chromosome.
The Stanford researchers are particularly excited about the prospects of the discovery since this kind of leukemia often has a grim outcome for most patients.
Professor Ravi Majeti and his team cheap nike air max 97 , whose findings are described in a paper in the latest issue of Proceedings of the National Academy of Sciences published on Monday, claimed the discovery was done almost by chance after they collected the type of leukemia cells from a patient.
"We were trying to keep these cells alive by providing them with different kinds of nutrients and growth factors," Majeti told Xinhua. "And we saw that sometimes, they shifted to white blood cells cheap air max 97 shoes , also known as macrophages. Instead of ignoring this fact, we decided to investigate what was making them change."
Majeti and his team were at odds to find the reason for the change until one researcher remembered an earlier study which showed that early B-cell mouse progenitor cells could be forced to become macrophages when exposed to certain transcription factors.
This meant that the main cause of the metamorphosis was a cocktail of proteins that bind to certain DNA sequences, turning the dangerous cells into macrophages that worked like "hound dogs" to track and kill cancer cells.
These leukemia cells not only turned into white blood cells, which are used by the immune system to fight all kinds of pathogens. They actually found the cancer cells and eliminated them by following their chemical trace.
Other treatments like chemotherapy also stimulate the immune system to track and destroy cancer cells cheap air max 97 , Majeti said, "but we have managed to find a way to actually turn the bad guys into good guys."
Reprogramming cancer cells to turn into immune cells can be a new strategy to
treat leukemia, but Majeti warned that it can take years to develop
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